诱导拓扑

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The proof is given that the category of linear fuzzy neighborhood spaces in the sense of Katsaras is isomorphic to that of co-towers of topological vector spaces and the intersection of the category of linear fuzzy neighborhood spaces with the category of type fuzzy topological vector spaces is the extract category of induced fuzzy topological vector spaces.

证明了Katsaras的线性fuzzy邻域空间范畴与分明线性拓扑空间的余塔空间范畴同构，而Katsaras的线性fuzzy邻域空间范畴与型fuzzy拓扑线性空间范畴的交集恰为诱导的fuzzy拓扑线性空间范畴。

It is pointed out that the category of L-fuzzy topological vector spaces is isomorphic to the full subcategory of the category of I -fuzzy topological vector spaces, whose elements are induced I -fuzzy topological spaces.

指出了格值模糊拓扑线性空间范畴同构于I型fuzzy线性拓扑空间范畴中诱导空间构成的满子范畴。

We prove that in every induced locally compact 〓fuzzy topological groupthere exist at least one regular fuzzy set-valued Haar measure.We define the fuzzymeasurable group and prove that the fuzzy set-valued Haar measure in a fuzzymeasurable group is essentially unique.We also construct the fuzzy topologicalstructure on a group by the fuzzy set-valued measure such that make it a fuzzytopological group.

证明了诱导局部紧〓模糊拓扑群上存在模糊集值Haar测度，定义了模糊可测群，通过对模糊可测群的研究证明了诱导局部紧〓模糊拓扑群上的模糊集值Haar测度本质上的唯一性，并利用模糊可测群上的模糊集值测度构造了群上的模糊拓扑结构使之成为模糊拓扑群。

Some important properties of L-topological spaces, induced topological spaces, fuzzy real lines and fuzzy metric spaces in topology on lattices are obtained.

在格上拓扑中，对L-拓扑空间、诱导拓扑空间、fuzzy 实直线和fuzzy度量空间等的性质研究，得到了一系列重要的结果。

The relation between the completeness of several local convex topology in normed vector space and that of induction topology of its unit ball was pointed out in this paper.

本文指出了赋范线性空间上的一些局部凸拓扑的完备性与它的单位球上相应的诱导拓扑的完备性之间的关系。

Studies also indicated that genistein exsists not only in soybeans but also in many kinds of plants such as vegetables and fruits.As a potential anticancer agent, the actional mechanisms of genistein mainly includes as follows:First, genistein can depress the activity of protein tyrosine kinase and transduction pathways for the phosphorylation of receptors and mitosis signal. So genistein can lead to cells' proliferation depressed. Second, genistein has minimal effects of phytoestrogens. It can be combined with estrogen receptor and improve the synthesis of cellular sex hormone binding glulobin, and improve the activity of UDP-glucuronyl transferase. Through these pathways, it can inhibit the cell activity of breast cancer and prostate cancer.

作为一个很有潜力的抗肿瘤物质，三羟异黄酮的作用机制主要包括：①抑制蛋白酪氨酸激酶活性，可阻抑PTK引起的受体磷酸化和有丝分裂的信号传递，导致癌细胞增殖受抑；②弱雌激素作用：可通过与体内雌激素受体结合，并可增加细胞内性激素结合球蛋白的合成，增加UDP-葡糖醛酸转移酶的活性等途径抑制乳腺癌和前列腺癌细胞活性；③拓扑酶Ⅰ和Ⅱ抑制剂，抑制细胞活性；④上调细胞周期性负性调节因子P21WAF1/CIP1的表达，使之负性调节因子作用增强；⑤可阻止胰岛素样生长因子-Ⅰ、肝细胞生长因子和神经生长因子的作用而抑制肿瘤生长；⑥其他：抗氧化作用、抑制热休克蛋白、诱导细胞凋亡、抑制新生血管生成和抑制多种耐药相关蛋白等。

Majority of acute leukemias in infant, either acute lymphoblastic leukemia or acute myeloblastic leukemia, posses a chromosomal translocation affecting the 11q23 chromosome region which specifically inoles the mixed-lineage leukemia gene.1-3 Most pediatric leukemias with MLL rearrangement clearly hae a remarkably short latency.1,4 MLL gene rearrangement is also associated with secondary leukemias of patients preiously treated with the topoisomerase II inhibitors.4 The latency of these secondary leukemias is similarly ery short.4 Of note, the concordance rate of leukemia with MLL rearrangement in infant monozygotic twins approximates to 100%,1,4 and identical breakpoint in the MLL gene was shared in these pairs of identical twin infants with concordant ALL.1,4 Moreoer, the unique and clonotypic MLL fusion gene was detectable in neonatal blood spots for Guthrie cards from non-twined indiiduals who subsequently deeloped ALL.1,4 These obserations indicate not only that MLL fusion is generated in utero but also that MLL fusion proteins could be capable of inducing leukemic transformation with few, if any, secondary mutations.2,3,4 Greaes et al speculate that an MLL fusion protein somehow promotes rapid transition to full-blown disease in patients ia ery rapid clonal expansion, genetic instability, or inhibition of DNA damage repair.4 In general, for clonal expansion of malignancies, tumor cells often hae acquired strategies that escape immune sureillance of the hosts.5,6 Immune escape mechanisms also contribute to the failure of graft-ersus-leukemia effect after allogeneic hematopoietic stem cell transplantation.7 Therefore, leukemia cells could acquire some immune escape mechanisms during leukemogenesis.

绪论 绝大多数的婴儿白血病，不管是急性淋巴性白血病或是急性骨髓性白血病，在染色体11q23部位有染色体易位的情况；这个部位的染色体易位牵连了混合谱系白血病基因。大多数具有MLL基因重排的儿童白血病潜伏期明显短很多。MLL基因重排也和经拓扑异构酶II抑制剂治疗后的继发性白血病有关。这些继发性白血病的潜伏期类似地都非常的短。很重要的是，单卵双胞胎婴儿同时患有或同时免于MLL基因重排阳性的白血病的一致性接近100%；并且同样患有ALL的同卵双胞胎的MLL基因的断裂点是一致的。而且，这种独特的克隆特异性的MLL融合基因能够从那些得ALL的非双生个体出生时的血斑标本中检测到。这些发现表明MLL融合基因产生在胎儿还在子宫的是后，而且MLL融合蛋白能过和其他的基因突变一起诱导白血病的产生。Greaes 等推测MLL融合蛋白在某种情况下同过快速克隆增殖，遗传的不稳定性或是DNA损伤修复的抑制促使疾病迅速地全面爆发。恶性肿瘤细胞的克隆增殖通常已经获得了逃避机体免疫监视的能力。免疫逃避机制也归因于异体外周血干细胞移植后移植物抗白血病作用的失效。所以，白血病细胞在白血病的产生过程中可能获得了某些免疫逃脱机制。

Metric molecular lattices and whose morphism are pointwise quasi-uniform continuous generalized order-homomorphism, which is called the cat.egory of pointwise p.q.

另外，每个由scott开集构成的拓扑也可由这种S－拟一致结构所诱导，恃别地，SCOt.t拓扑可由这种S－拟一致结构所诱导。

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induced homomorphism：导出同态

induced fiber bundle 诱导纤维丛 | induced homomorphism 导出同态 | induced topology 诱导拓扑

induced homomorphism：诱导同态

诱导特征[标]|induced character | 诱导同态|induced homomorphism | 诱导拓扑|induced topology

induced topology：诱导拓扑

induced homomorphism 导出同态 | induced topology 诱导拓扑 | induction 归纳

induzierte Topologie induced topology：诱导拓扑

Induktionsvoraussetzung induction hypothesis 归纳假设 | induzierte Topologie induced topology 诱导拓扑 | Infimum infimum 最大下界

Induktionsvoraussetzung induction hypothesis：归纳假设

Induktionsverankerung base step 基本步骤 | Induktionsvoraussetzung induction hypothesis 归纳假设 | induzierte Topologie induced topology 诱导拓扑

Infimum infimum：最大下界

induzierte Topologie induced topology 诱导拓扑 | Infimum infimum 最大下界 | Inhomogenitaet inhomogeneity 不均匀性

induced subgraph：诱导子图

诱导拓扑|induced topology | 诱导子图|induced subgraph | 余标架|coframe